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This paradox highlights the stage-dependent duality of AMPK signaling: adaptive in early disease but maladaptive in later stages, as documented in recent clinical evidence (Wang et al., 2009)

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The maturity of these factors is then facilitated by erinacines and cordycepin, which maintain neurotrophic levels by increasing both pro-BDNF and BDNF through established mechanisms of synthesis and release [14,95]